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SVIL promotes ovarian cancer progression and epithelial-mesenchymal transition under hypoxic conditions through the TGF-β/Smad pathway.

Chenxue QiMengmeng LyuZhipeng YaoFan ZhangYan ZhuMin QiXiaoxiang CaoCongzhu LiHongping XiaDongfang Dai
Published in: Gynecologic oncology (2024)
SVIL highly expressed in the hypoxic microenvironment can increase ovarian cancer progression and cisplatin resistance by activating TGFβ1/smad2/3 pathway. Our study demonstrated that SVIL may be a novel target for the treatment of ovarian cancer.
Keyphrases
  • transforming growth factor
  • epithelial mesenchymal transition
  • signaling pathway
  • stem cells
  • combination therapy