Neuronal Alterations in Secondary Thalamic Degeneration Due to Cerebral Infarction: A 11 C-Flumazenil Positron Emission Tomography Study.

Secondary thalamic neuronal damage may manifest as a decrease in central benzodiazepine receptors in patients with cerebral infarction and internal carotid artery or middle cerebral artery disease, which may be associated with frontal lobe dysfunction.