Male sickle cell patients, compensated transpubertal hypogonadism and normal final growth.

Paulo Roberto Juliano MartinsFernanda Bernadelli De VitoGláucia Aparecida Domingos ResendeJosé KerbauyGilberto de Araújo PereiraHelio Moraes-Souza Maria Stella FigueiredoIeda Therezinha Verreschi

Published in: Clinical endocrinology (2019)

These data, in addition to both the clinical and biochemical signs of hypoandrogenism associated with normal to elevated T levels strongly suggest a peripheral origin of hypogonadism, which is probably due to androgen resistance in the patients with SCD.

Keyphrases

end stage renal disease
ejection fraction
newly diagnosed
chronic kidney disease
replacement therapy
peritoneal dialysis
electronic health record
patient reported outcomes
data analysis