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Essential Role Of High Glucose-Induced Overexpression Of PKCβ And PKCδ In GLP-1 Resistance In Rodent Cardiomyocytes.

Xietian PanJiangwei ChenTingting WangMingming ZhangHaichang WangHaokao Gao
Published in: Diabetes, metabolic syndrome and obesity : targets and therapy (2019)
Our study found out a new mechanism of GLP-1 resistance that high glucose-induced overexpression of PKCβ and PKCδ impaired cardioprotective effects of GLP-1 by downregulation of GLP-1R and inhibition of GLP-1 post-receptor anti-apoptotic signaling pathways, thus provided a new perspective in treating myocardial IRI in diabetic patients.
Keyphrases
  • high glucose
  • endothelial cells
  • cell proliferation
  • signaling pathway
  • protein kinase
  • cell death
  • transcription factor
  • oxidative stress
  • diabetic rats
  • endoplasmic reticulum stress
  • drug induced