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Impairment of AMPK-α2 augments detrusor contractions in bladder ischemia.

Jing-Hua YangWanting NiuYedan LiKazem M Azadzoi
Published in: Investigative and clinical urology (2021)
Ischemia induces post-translational modification of AMPK-α2 protein. Impairment of AMPK-α2 may contribute to overactive detrusor contractions and loss of nerve fibers in bladder ischemia. AMPK activators may have therapeutic potential against detrusor overactivity and neurodegeneration in bladder conditions involving ischemia.
Keyphrases
  • skeletal muscle
  • botulinum toxin
  • spinal cord injury
  • protein kinase