Association between subarachnoid hemorrhage-induced hydrocephalus and hydromyelia: pathophysiological changes developed in an experimental model.

Subependymal basement membrane destruction, blood cell accumulation on it, ependymal cell desquamation, increased cerebrospinal fluid (CSF) secretion, and increased ICP in the central channel that causes hydromyelia. When these pathological changes are chronically apparent, they may reflect on CSF pathways and cause permanent SAIH. Preventing long-time SAH-induced hydromyelia is believed to reduce the high rate of treatment-requiring SAIH.