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Impact of glucagon response on early postprandial glucose excursions irrespective of residual β-cell function in type 1 diabetes: A cross-sectional study using a mixed meal tolerance test.

Ayako ItoIchiro HorieMasaki MiwaAyaka SakoTetsuro NiriYomi NakashimaRiyoko ShigenoAi HaraguchiShoko NatsudaSatoru AkazawaAkie KamadaAtsushi KawakamiNorio Abiru
Published in: Journal of diabetes investigation (2021)
The dysregulated glucagon likely contributes to postprandial hyperglycemia independent of the residual β-cell functions during the progression of type 1 diabetes.
Keyphrases
  • blood glucose
  • type diabetes
  • glycemic control
  • single cell
  • cell therapy
  • cardiovascular disease
  • insulin resistance
  • diabetic rats
  • adipose tissue