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Remnant Pancreas Volume Affects New-Onset Impaired Glucose Homeostasis Secondary to Pancreatic Cancer.

Jie YangChunlu TanYa LiuZhenjiang ZhengXubao LiuYonghua Chen
Published in: Biomedicines (2024)
Reduced RPV causing beta-cell dysfunction might be one of the mechanisms of NOD secondary to PDAC. Subjects with sufficient pancreas volume could not be detected earlier when regarding patients with NOD as the population at risk for PDAC.
Keyphrases
  • single cell
  • cell therapy
  • innate immune
  • blood glucose
  • stem cells
  • adipose tissue
  • blood pressure
  • skeletal muscle
  • bone marrow
  • insulin resistance
  • glycemic control