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Axonal distribution of mitochondria maintains neuronal autophagy during aging via eIF2β.

Kanako ShinnoYuri MiuraKoichi M IijimaEmiko SuzukiKanae Ando
Published in: bioRxiv : the preprint server for biology (2024)
Loss of axonal mitochondria impairs autophagy and accumulates proteins in the axonLoss of axonal mitochondria upregulates eIF2β and downregulates p-eIF2αNeuronal upregulation of eIF2β induces autophagic defects and locomotor dysfunctionLowering eIF2β rescues autophagic defects caused by loss of axonal mitochondria.
Keyphrases
  • cell death
  • spinal cord injury
  • reactive oxygen species
  • endoplasmic reticulum
  • oxidative stress
  • endoplasmic reticulum stress
  • cell proliferation
  • cerebral ischemia
  • mouse model
  • blood brain barrier