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Advanced Glycation End Products Enhance Murine Monocyte Proliferation in Bone Marrow and Prime Them into an Inflammatory Phenotype through MAPK Signaling.

Xian JinLiang LiuYaping ZhangYin XiangGuizhi YinYi LuLudong ShiJian DongChengxing Shen
Published in: Journal of diabetes research (2018)
AGEs promote bone marrow monocytes to proliferate and drive them into an inflammatory phenotype through p38 and ERK activation.
Keyphrases
  • immune response
  • bone marrow
  • dendritic cells
  • signaling pathway
  • oxidative stress
  • mesenchymal stem cells
  • pi k akt
  • peripheral blood
  • cell proliferation
  • endothelial cells