The Constitutive Lack of α7 Nicotinic Receptor Leads to Metabolic Disorders in Mouse.
Blandine GausserèsJunjun LiuEwout FoppenCécile Tourrel-CuzinAna Rodriguez Sanchez-ArchidonaEtienne DelangreCéline Cruciani-GuglielmacciStéphanie PonsUwe MaskosBernard ThorensChristophe MagnanJamileh MovassatKamel MaouchePublished in: Biomolecules (2020)
Our work highlights the important role of α7 nAChR in glucose homeostasis. The constitutive lack of α7 nAChR suggests a novel pathway influencing the pathogenesis of T2D.