LECT2 modulates dendritic cell function after H. pylori infection via the CD209a receptor.
Xiaofen ZhangKefang SunChenxi TangLi CenSha LiWei ZhuPeihao LiuYishu ChenChaohui YuLan LiPublished in: Journal of gastroenterology and hepatology (2023)
This study reveals that LECT2 modulated the functions of H. pylori-primed DCs in a CD209a-dependent manner, which might hinder the clearance of H. pylori and contribute to its colonization.