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CCN4 (WISP-1) reduces apoptosis and atherosclerotic plaque burden in an ApoE mouse model.

Helen WilliamsSteven SimmondsAndrew BondAlexandros SomosZe LiTessa ForbesRosaria BiancoCelyn DugdaleZoe BrownHelen RiceAndrew HermanJason JohnsonSarah George
Published in: Atherosclerosis (2024)
We conclude that in an atherosclerosis model the most important action of CCN4 is the effect on cell apoptosis. CCN4 provides pro-survival signals and leads to reduced cell death, lower macrophage number, smaller lipid core size and reduced atherosclerotic plaque burden. As such, the pro-survival effect of CCN4 is worthy of further investigation, in a bid to find a therapeutic for atherosclerosis.
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