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Carbon Monoxide Activates PERK-Regulated Autophagy to Induce Immunometabolic Reprogramming and Boost Antitumor T-cell Function.

Paramita ChakrabortyRasesh Y ParikhSeungho ChoiDanh TranMonika GoozZachariah T HedleyDo-Sung KimDariusz PytelInhong KangSatish N NadigGyda C BeesonLauren E BallMeenal MehrotraHongjun WangStefano BertoViswanathan PalanisamyHong LiShilpak ChatterjeePaulo C RodriguezEduardo N MaldonadoJ Alan DiehlVamsi K GangarajuShikhar Mehrotra
Published in: Cancer research (2022)
Transient activation of ER stress with carbon monoxide drives mitochondrial biogenesis and protective autophagy that elicits superior antitumor T-cell function, revealing an approach to improving adoptive cell efficacy therapy.
Keyphrases
  • endoplasmic reticulum stress
  • cell therapy
  • oxidative stress
  • cell death
  • signaling pathway
  • single cell
  • transcription factor
  • stem cells
  • mesenchymal stem cells
  • brain injury
  • subarachnoid hemorrhage
  • replacement therapy