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Aconitine induces cardiomyocyte damage by mitigating BNIP3-dependent mitophagy and the TNFα-NLRP3 signalling axis.

Fu PengNan ZhangChunting WangXiaoyun WangWei HuangCheng PengGu HeBo Han
Published in: Cell proliferation (2019)
Our findings lay a foundation for the application of a TNFα inhibitor and BNIP3 to aconitine-induced cardiac toxicity prevention and therapy, thereby demonstrating potential for further investigation.
Keyphrases
  • high glucose
  • rheumatoid arthritis
  • oxidative stress
  • diabetic rats
  • nlrp inflammasome
  • endothelial cells
  • left ventricular
  • angiotensin ii
  • drug induced
  • heart failure
  • risk assessment
  • stress induced
  • oxide nanoparticles