Telomere Shortening in Hypertensive Heart Disease Depends on Oxidative DNA Damage and Predicts Impaired Recovery of Cardiac Function in Heart Failure.
Moritz BrandtHendrik DörschmannSana'a KhraisatTanja KnoppJulia RingenSanela KalinovicVenkata GarlapatiSvenja SiemerMichael MolitorSebastian GöbelRoland StauberSusanne Helena KarbachThomas MunzelAndreas DaiberPhilip WenzelPublished in: Hypertension (Dallas, Tex. : 1979) (2022)
Cardiomyocyte telomere shortening and oxidative damage in heart failure with reduced ejection fraction induced by excess neurohormonal activation depends on NOX2-derived superoxide and may help to stratify HF therapy.