These studies of normal tissues have highlighted the specific drivers of clonal expansion and how frequently they appear in us. Their high prevalence throughout cancer-free individuals necessitates reconsideration of the oncogenicity of these mutations, which could shape methods of detection, prevention and treatment of cancer, as well as of the potential impact of these mutations on tissue function and our health.
Keyphrases
- papillary thyroid
- squamous cell
- gene expression
- induced apoptosis
- public health
- healthcare
- risk factors
- mental health
- lymph node metastasis
- cell cycle arrest
- squamous cell carcinoma
- health information
- signaling pathway
- risk assessment
- climate change
- social media
- endoplasmic reticulum stress
- loop mediated isothermal amplification
- combination therapy