Deficiency of Antioxidative Paraoxonase 2 (Pon2) Leads to Increased Number of Phenotypic LT-HSCs and Disturbed Erythropoiesis.
Lisa SpieckerInes WitteJulia MehligViral ShahMarkus MeyerhöferPatricia S HaehnelVictoria PetermannAndrea SchülerPiyush MoreNina Cabezas-WallscheidSven HorkeAndrea PautzAndreas DaiberDaniel SascaThomas KindlerHartmut KleinertPublished in: Oxidative medicine and cellular longevity (2021)
In summary, our current data indicate that PON2 is involved in the regulation of HSC functions.