The HIV co-receptor CCR5 regulates osteoclast function.
Ji-Won LeeAkiyoshi HoshinoKazuki InoueTakashi SaitouShunsuke UeharaYasuhiro KobayashiSatoshi UehaKouji MatsushimaAkira YamaguchiYuuki ImaiTadahiro IimuraPublished in: Nature communications (2017)
C-C chemokine receptor 5 (CCR5) is a co-receptor of HIV. Epidemiological findings suggest that the functional loss of CCR5 is correlated with a lower incidence of bone-destructive diseases as well as of HIV transmission. However, it is not clear whether CCR5 is involved in regulation of the function of bone cells, in addition to that of immune cells. Here we show that blockade of CCR5 using specific antibodies impairs human osteoclast function in vitro. Ccr5-deficient (Ccr5 -/- ) mice presented with dysfunctional osteoclasts and were resistant to osteoporosis induced by receptor activator of nuclear factor kappa-B ligand (RANKL), which triggers osteoporosis independently of inflammatory and immunomodulatory pathways. Furthermore, Ccr5 deficiency impairs the cellular locomotion and bone-resorption activity of osteoclasts, which is associated with the disarrangement of podosomes and adhesion complex molecules including Pyk2. Overall, the data provides evidence that CCR5 has an essential role in bone-destructive conditions through the functional regulation of osteoclasts.
Keyphrases
- bone loss
- nuclear factor
- dendritic cells
- regulatory t cells
- bone mineral density
- antiretroviral therapy
- hiv positive
- hiv infected
- toll like receptor
- postmenopausal women
- human immunodeficiency virus
- hepatitis c virus
- hiv aids
- hiv testing
- endothelial cells
- type diabetes
- body composition
- soft tissue
- cell death
- oxidative stress
- pseudomonas aeruginosa
- skeletal muscle
- cystic fibrosis
- big data
- insulin resistance
- artificial intelligence
- candida albicans
- biofilm formation