Impact of lipopolysaccharide-induced acute inflammation on baroreflex-controlled sympathetic arterial pressure regulation.

Takeshi TohyamaKeita SakuToru KawadaTakuya KishiKeimei YoshidaTakuya NishikawaHiroshi MannojiKazuhiro KamadaKenji SunagawaHiroyuki Tsutsui

Published in: PloS one (2018)

LPS-induced acute inflammation markedly increased SNA via resetting of the baroreflex neural arc, and suppressed the peripheral arc. The balance between the augmented neural arc and suppressed peripheral arc determines SNA and hemodynamics in LPS-induced acute inflammation.

Keyphrases

lipopolysaccharide induced
inflammatory response
oxidative stress
liver failure
anti inflammatory
hepatitis b virus
acute respiratory distress syndrome
extracorporeal membrane oxygenation
mechanical ventilation