IL-22Ra1 is induced during influenza infection by direct and indirect TLR3 induction of STAT1.

K D HebertN MclaughlinZhe ZhangA CiprianiJ F AlcornDerek A Pociask

Published in: Respiratory research (2019)

Collectively, these data suggest epithelial cells may optimize the beneficial effects of IL-22 through the induction of the IL-22 receptor during viral infection in the lung.

Keyphrases

rheumatoid arthritis
immune response
toll like receptor
cell proliferation
oxidative stress
systemic lupus erythematosus
deep learning
nuclear factor
artificial intelligence