Login / Signup

Type 2 Diabetes Modifies the Association of CAD Genomic Risk Variants With Subclinical Atherosclerosis.

Natalie R HasbaniKenneth E WestermanSoo Heon KwakHan-Yang ChenXihao LiDaniel A DiCorpoJennifer WesselJoshua C BisChloe SarnowskiPeitao WuLawrence F BielakXiuqing GuoNancy Heard- CostaGregory L KinneyMichael C MahaneyMay E MontasserNicholette D D AllredLaura M RaffieldJames G TerryLisa R YanekJessica BonDonald W BowdenJennifer A BrodyRavindranath DuggiralaDavid R JacobsRita R KalyaniLeslie A LangeBraxton D MitchellJennifer A SmithKent D TaylorApril P CarsonJoanne E CurranMyriam FornageBarry I FreedmanStacey GabrielRichard A GibbsNamrata GuptaSharon L R KardiaBrian G KralZeineen MominAnne B NewmanWendy S PostKarine A Viaud-MartinezKendra A YoungLewis C BeckerAlain G BertoniJohn E BlangeroJohn Jeffrey CarrKatherine PratteBruce M PsatyStephen S Richnull nullJoseph C WuRajeev MalhotraPatricia A PeyserAlanna C MorrisonRamachandran S VasanXihong LinJerome I RotterJames B MeigsAlisa K ManningPaul S de Vries
Published in: Circulation. Genomic and precision medicine (2023)
These results highlight T2D as an important modifier of rare variant associations in CAD loci with CAC.
Keyphrases
  • coronary artery disease
  • type diabetes
  • copy number
  • cardiovascular disease
  • genome wide
  • glycemic control
  • genome wide association study
  • insulin resistance