Red Blood Cell and Endothelial eNOS Independently Regulate Circulating Nitric Oxide Metabolites and Blood Pressure.
Francesca LeoTatsiana SuvoravaSophia K HeuserJunjie LiAnthea LoBueFrederik BarbarinoEugenia PiragineRebekka SchneckmannBeate HutzlerMiranda E GoodBernadette O FernandezLukas VornholzStephen Colin RogersAllan DoctorMaria GrandochJohannes StegbauerEddie WeitzbergMartin FeelischJon O LundbergBrant E IsaksonMalte KelmMiriam M Cortese-KrottPublished in: Circulation (2021)
These data reveal that eNOS in ECs and RBCs contribute independently to blood pressure homeostasis.
Keyphrases
- blood pressure
- red blood cell
- nitric oxide synthase
- nitric oxide
- endothelial cells
- hypertensive patients
- pi k akt
- heart rate
- ms ms
- genome wide
- hydrogen peroxide
- big data
- electronic health record
- type diabetes
- signaling pathway
- metabolic syndrome
- adipose tissue
- gene expression
- machine learning
- skeletal muscle
- data analysis
- weight loss
- deep learning
- glycemic control