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Chronic Diclofenac Exposure Increases Mitochondrial Oxidative Stress, Inflammatory Mediators, and Cardiac Dysfunction.

Phung N ThaiLu RenWilson XuJames OvertonValeriy TimofeyevCarol E NaderMichael HaddadJun YangAldrin V GomesBruce D HammockNipavan ChiamvimonvatPadmini Sirish
Published in: Cardiovascular drugs and therapy (2021)
Together, chronic treatment with DIC resulted in severe cardiotoxicity, which was mediated, in part, by an increase in mitochondrial oxidative stress.
Keyphrases
  • oxidative stress
  • diabetic rats
  • ischemia reperfusion injury
  • dna damage
  • induced apoptosis