Helicobacter pylori infection modulates endogenous hydrogen sulfide production in gastric cancer AGS cells.

Yoshinari KawaharaYuka HirashitaChikako TamuraYoko KudoKumiko SakaiKazumi TogoKensuke FukudaOsamu MatsunariKazuhisa OkamotoRyo OgawaKazuhiro MizukamiTadayoshi OkimotoMasaaki KodamaKazunari Murakami

Published in: Helicobacter (2020)

H pylori infection modulates endogenous H2 S production in AGS cells, suggesting that H2 S might be one of the bioactive molecules involved in the biological mechanisms of gastric mucosal disease including mucosal atrophy.

Keyphrases

induced apoptosis
helicobacter pylori infection
cell cycle arrest
endoplasmic reticulum stress
ulcerative colitis
oxidative stress