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Gene-environment interactions: Epstein-Barr virus infection and risk of pediatric-onset multiple sclerosis.

Amin ZiaeiOlivia SolomonT Charles CasperMichael WaltzBianca Weinstock-GuttmanGreg AaenYolanda WheelerJennifer GravesLeslie BensonMark GormanMary R RenselSoe MarTim LotzeBenjamin M GreenbergTanuja ChitinisAmy T WaldmanLauren B KruppJudith A JamesJanace HartLisa F BarcellosEmmanuelle Waubant
Published in: Multiple sclerosis (Houndmills, Basingstoke, England) (2024)
POMS risk variant. Our results suggest an important role of antigen-presenting cells (APCs) in EBV-associated POMS risk.
Keyphrases
  • epstein barr virus
  • multiple sclerosis
  • diffuse large b cell lymphoma
  • induced apoptosis
  • cell cycle arrest
  • oxidative stress
  • gene expression
  • cell proliferation
  • endoplasmic reticulum stress
  • childhood cancer