An alternative way of SARS-COV-2 to induce cell stress and elevated DNA damage risk in cardiomyocytes without direct infection.
Houqing ZhouXiaohu RenYang YangBenhong XuYichong LiYin FengFang ShisongJian-Jun LiuPublished in: Immunity, inflammation and disease (2022)
These findings indicated the possibilities of SARS-CoV-2 inducing stress and elevating DNA damage risk to cardiomyocytes without direct infection.