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Melatonin inhibits the proliferation of breast cancer cells induced by bisphenol A via targeting estrogen receptor-related pathways.

Tianjiao WangBowen LiuYanan GuanMiaomiao GongWeiying ZhangJinjin PanYanan LiuRui LiangYuhui YuanLihong Ye
Published in: Thoracic cancer (2018)
Conclusions: Therefore, these results demonstrated that melatonin could abrogate BPA-induced proliferation of BC cells. Therapeutically, melatonin could be regarded as a potential medication for BPA-associated BC.
Keyphrases
  • estrogen receptor
  • signaling pathway
  • breast cancer cells
  • induced apoptosis
  • cell cycle arrest
  • high glucose
  • healthcare
  • diabetic rats
  • drug induced
  • emergency department
  • cancer therapy
  • cell death
  • pi k akt
  • adverse drug