Histopathological evaluation of the lungs in experimental autoimmune encephalomyelitis.
Sungmoo HongJeongtae KimKyungsook JungMee Jung AhnChangjong MoonYoshihiro NomuraHiroshi MatsudaAkane TanakaHyohoon JeongTaekyun ShinPublished in: Journal of veterinary science (2024)
Taken together, these findings suggest that the increased OPN level in lungs of EAE mice led to inflammation; concurrent increases in proinflammatory factors (OPN and galectin-3) caused pulmonary impairment.