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PPARγ ligands increase antileukemic activity of second- and third-generation tyrosine kinase inhibitors in chronic myeloid leukemia cells.

E Glodkowska-MrowkaA Manda-HandzlikA Stelmaszczyk-EmmelI SeferynskaT StoklosaJ PrzybylskiPiotr Mrówka
Published in: Blood cancer journal (2016)
Keyphrases
  • chronic myeloid leukemia
  • induced apoptosis
  • cell cycle arrest
  • insulin resistance
  • endoplasmic reticulum stress
  • oxidative stress
  • cell death
  • metabolic syndrome
  • adipose tissue