Post-reperfusion syndrome (PRS) is a severe and highly lethal syndrome that occurs after declamping the portal vein forceps during liver transplantation. It is marked by severe hemodynamic disturbances manifested by decreased mean arterial pressure, increased heart rate and elevated pulmonary artery pressure. The complex pathogenesis of PRS remains understudied. It is generally believed to be related to the large amount of acidic, cold blood that enters the circulation after release of the portal clamp. This blood is rich in oxygen-free radicals and metabolic toxins, which not only aggravate the ischemia-reperfusion injury of the liver but also further attack the systemic organs indiscriminately. Considering the range of possible adverse prognoses including acute kidney injury, delirium and graft nonfunction, it is imperative that clinicians increase their awareness and prevention of PRS. The aim of this article is to review the current risk factors, pathophysiological mechanisms and prevention strategies for PRS.
Keyphrases
- risk factors
- heart rate
- pulmonary artery
- acute kidney injury
- ischemia reperfusion injury
- coronary artery
- pulmonary hypertension
- heart rate variability
- case report
- blood pressure
- cardiac surgery
- acute myocardial infarction
- early onset
- cerebral ischemia
- heart failure
- oxidative stress
- drug induced
- palliative care
- ionic liquid
- left ventricular
- blood brain barrier
- percutaneous coronary intervention
- electronic health record
- subarachnoid hemorrhage