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Inflammasome-Independent Mechanism of NLRP3 Is Critical for Platelet GPIb-IX Function and Thrombosis.

Xiaoyan ChenJingke LiPu LiuYangfan ZhouTongtong ZhangLi LiJingqi ShiXin DengYilin ShengWei ChenDi WangHu Hu
Published in: Thrombosis and haemostasis (2024)
 NLRP3 promoted the function of the major platelet adhesion receptor GPIb-IX without involving NLRP3 inflammasome assembly or IL-1β production.
Keyphrases
  • nlrp inflammasome
  • pseudomonas aeruginosa
  • binding protein
  • cell migration