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Preclinical study in a postoperative pain model to investigate the action of ketamine, lidocaine, and ascorbic acid in reversing fentanyl-induced, non-glutamate-dependent hyperalgesia.

Marina Ayres DelgadoLuana Assis FerreiraBianka Jaciara Dos Santos GomesIsis Katarine Orlandi LeiteMarcus Vinícius GomezCélio Castro-Junior
Published in: Pain reports (2023)
Fentanyl induced a hyperalgesic effect that last few days in a postoperative model of pain. Hyperalgesic effect was not totally inhibited by ketamine and lidocaine in rats. Increased glutamate release was not the main molecular mechanism of fentanyl-induced hyperalgesia.
Keyphrases
  • high glucose
  • diabetic rats
  • postoperative pain
  • neuropathic pain
  • pain management
  • drug induced
  • chronic pain
  • stem cells
  • oxidative stress
  • spinal cord
  • spinal cord injury
  • bone marrow