Grey Matter Atrophy and its Relationship with White Matter Lesions in Patients with Myelin Oligodendrocyte Glycoprotein Antibody-associated Disease, Aquaporin-4 Antibody-Positive Neuromyelitis Optica Spectrum Disorder, and Multiple Sclerosis.
Rosa CorteseMarco BattagliniFerran PradosGiordano GentileLudovico LuchettiAlessia BianchiLukas HaiderAnu JacobJacqueline PalaceSilvia MessinaFriedemann PaulRomain MarignierFrançoise Durand-DubiefCarolina de Medeiros RimkusSamira Luisa Apostolos PereiraDouglas Kazutoshi SatoMassimo FilippiMaria Assunta RoccaLaura CacciaguerraÀlex RoviraJaume Sastre-GarrigaGeorgina ArrambideYaou LiuYunyun DuanClaudio GasperiniCarla TortorellaSerena RuggieriMaria Pia AmatoMonica UlivelliSergiu GroppaMatthias GrotheSara LlufriuMaria SepulvedaCarsten LukasBarbara BellenbergRuth SchneiderPiotr SowaElisabeth G CeliusAnne-Katrin PröbstelCristina GranzieraÖzgür YaldizliJannis MüllerBruno StankoffBenedetta BodiniFrederik BarkhofOlga CiccarelliNicola De Stefanonull nullPublished in: Annals of neurology (2024)
GM atrophy is more widespread in RRMS compared with the other two conditions. MOGAD primarily affects the temporal cortex, whereas AQP4+NMOSD mainly involves the occipital cortex. In MOGAD and RRMS, lesion-related tract degeneration is associated with atrophy, but this link is absent in AQP4+NMOSD. ANN NEUROL 2024;96:276-288.