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Intestinal epithelia and myeloid immune cells shape colitis severity and colorectal carcinogenesis via High-mobility group box protein 1.

Katharina FoelschPenelope PelczarElisabeth ZierzStephanie KondratowiczMinyue QiChristian MüllerMalik AlawiSina HuebenerTill ClauditzNicola GaglianiSamuel HuberPeter Huebener
Published in: Journal of Crohn's & colitis (2024)
Enterocytes and myeloid cells context-dependently regulate host responses to severe colitis and maladaptive intestinal wound healing via HMGB1.
Keyphrases
  • wound healing
  • induced apoptosis
  • dendritic cells
  • bone marrow
  • acute myeloid leukemia
  • binding protein
  • cell cycle arrest
  • ulcerative colitis
  • transcription factor
  • protein protein
  • oxidative stress
  • drug induced