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Natural Killer Group 2D Receptor and B1a Cells Crosstalk in Post-Hepatitis C Virus Infection Hepatocellular Carcinoma and Cirrhosis.

Reham HammadMona A EldosokyAlshaimaa M MosaadAsmaa M El-NasserFatma M KotbSalwa I ElshennawyNoha Abdel-Rahman EldesokyMohamed A SelimGina G NaguibOssama A AhmedMohamed AlboraieReda Badr Aglan
Published in: Journal of hepatocellular carcinoma (2022)
Downregulation of B1a frequency and NKG2D intensity is implicated in the progression of CHCV infection to cirrhosis and HCC. NKG2D receptor is associated with the frequency of circulating B1a cells. NKG2D intensity and B1a% can be used as indicators of CHCV progression.
Keyphrases
  • induced apoptosis
  • nk cells
  • cell cycle arrest
  • hepatitis c virus infection
  • natural killer cells
  • signaling pathway
  • cell proliferation
  • endoplasmic reticulum stress
  • oxidative stress