Acute hyperglycemia suppresses left ventricular diastolic function and inhibits autophagic flux in mice under prohypertrophic stimulation.
Jiahe XieKai CuiHuixin HaoYingxue ZhangHairuo LinZhenhuan ChenXiaobo HuangShiping CaoWangjun LiaoJianping BinMasafumi KitakazeYulin LiaoPublished in: Cardiovascular diabetology (2016)
Acute hyperglycemia suppresses diastolic function, damages mitochondrial energy signaling, and inhibits autophagic flux in prohypertrophic factor-stimulated cardiomyocytes.
Keyphrases
- left ventricular
- liver failure
- cell death
- respiratory failure
- blood pressure
- signaling pathway
- hypertrophic cardiomyopathy
- drug induced
- aortic dissection
- heart failure
- acute myocardial infarction
- cardiac resynchronization therapy
- aortic stenosis
- oxidative stress
- ejection fraction
- mitral valve
- diabetic rats
- type diabetes
- coronary artery disease
- extracorporeal membrane oxygenation
- intensive care unit
- high fat diet induced
- atrial fibrillation
- adipose tissue
- skeletal muscle
- transcatheter aortic valve replacement
- percutaneous coronary intervention
- mechanical ventilation
- insulin resistance