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Colocalized targeting of TGF-β and PD-L1 by bintrafusp alfa elicits distinct antitumor responses.

Yan LanTsz-Lun YeungHui HuangAnsgar A WegenerSomdutta SahaMira Toister-AchituvMolly H JenkinsLi-Ya ChiuAdam LazorchakOhad TarcicHong WangJin QiGeorge LockeDoron KalimiGuozhong QinBo MarelliHuakui YuAlec W GrossMelissa G DernerMaria SolovievMathieu BotteAroop SircarHong MaVanita D SoodDong ZhangFeng JiangKin-Ming Lo
Published in: Journal for immunotherapy of cancer (2022)
BA more effectively blocks TGF-β by targeting TGF-β trap to the tumor via PD-L1 binding. Such colocalized targeting elicits distinct and superior antitumor responses relative to single agent combination therapy.
Keyphrases
  • combination therapy
  • transforming growth factor
  • cancer therapy
  • epithelial mesenchymal transition
  • drug delivery
  • dna binding
  • transcription factor
  • binding protein