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PFKP deubiquitination and stabilization by USP5 activate aerobic glycolysis to promote triple-negative breast cancer progression.
Zi-Mei Peng
Xiao-Jian Han
Tao Wang
Jian-Jun Li
Chun-Xi Yang
Fang-Fang Tou
Zhen Zhang
Published in:
Breast cancer research : BCR (2024)
Our study established the USP5-PFKP axis as an important regulatory mechanism of TNBC progression and provided a rationale for future therapeutic interventions in the treatment of TNBC.
Keyphrases
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physical activity
clinical trial
transcription factor
current status
replacement therapy