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Antitumor Necrosis Factor-like Ligand 1A Therapy Targets Tissue Inflammation and Fibrosis Pathways and Reduces Gut Pathobionts in Ulcerative Colitis.

Mina Hassan-ZahraeeZhan YeLi XiMary Lynn BanieckiXingpeng LiCraig L HydeJenny ZhangNancy RahaFridrik KarlssonJie QuanDaniel ZiemekSrividya NeelakantanChristopher LepsyJessica R AllegrettiJacek RomatowskiEllen J ScherlMaria KlopockaSilvio DaneseDeepa E ChandraUwe SchoenbeckMichael S VincentRandy LongmanKenneth E Hung
Published in: Inflammatory bowel diseases (2021)
The ability of PF-06480605 to engage and inhibit colonic TL1A, targeting inflammatory T cell and fibrosis pathways, provides the first-in-human mechanistic data to guide anti-TL1A therapy for the treatment of IBD.
Keyphrases
  • ulcerative colitis
  • oxidative stress
  • endothelial cells
  • electronic health record
  • induced pluripotent stem cells
  • stem cells
  • machine learning
  • combination therapy
  • mesenchymal stem cells
  • replacement therapy