Maternal inflammation exacerbates neonatal hyperoxia-induced kidney injury in rat offspring.
Chung-Ming ChenHsiu-Chu ChouPublished in: Pediatric research (2019)
Maternal inflammation exacerbates neonatal hyperoxia-induced kidney injury and the underlying mechanism may be related to activation of TLR4 and increased oxidative stress.
Keyphrases
- oxidative stress
- diabetic rats
- high glucose
- dna damage
- immune response
- drug induced
- ischemia reperfusion injury
- toll like receptor
- inflammatory response
- induced apoptosis
- pregnancy outcomes
- high fat diet
- endothelial cells
- type diabetes
- physical activity
- nuclear factor
- weight loss
- pregnant women
- signaling pathway
- insulin resistance
- endoplasmic reticulum stress