Dapagliflozin-Induced Myocardial Flow Reserve Improvement is not Associated with HDL Ability to Stimulate Endothelial Nitric Oxide Production.

Umberto CapeceChiara PavanelloFrancesca CintiLucia LeccisottiTeresa MezzaGea CiccarelliSimona MoffaGianfranco Di GiuseppeLaura SoldovieriMichela BrunettiAlessandro GiordanoAndrea GiaccariLaura CalabresiAlice Ossoli

Published in: Diabetes therapy : research, treatment and education of diabetes and related disorders (2023)

EudraCT No. 2016-003614-27; ClinicalTrials.gov Identifier: NCT03313752.

Keyphrases

nitric oxide
high glucose
endothelial cells
diabetic rats
left ventricular
nitric oxide synthase
hydrogen peroxide
drug induced
oxidative stress
heart failure