Login / Signup

GTSE1 promotes nasopharyngeal carcinoma proliferation and angiogenesis by upregulating STMN1.

Jiadi DongJingjing ChenYidong WuJiangyu Yan
Published in: Cell division (2024)
GTSE1 was highly expressed in NPC, and silencing of GTSE1 ameliorated the malignant processes of NPC cells by upregulating STMN1, suggesting a possible therapeutical target for NPC.
Keyphrases
  • induced apoptosis
  • cell cycle arrest
  • signaling pathway
  • endothelial cells
  • vascular endothelial growth factor
  • endoplasmic reticulum stress
  • cell death
  • oxidative stress
  • pi k akt
  • cell proliferation