Login / Signup

Curcumin and a hemi-analogue with improved blood-brain barrier permeability protect against amyloid-beta toxicity in Caenorhabditis elegans via SKN-1/Nrf activation.

Elaine Hui-Chien LeeSherlyn Sheau-Chin LimKah-Hay YuenChong-Yew Lee
Published in: The Journal of pharmacy and pharmacology (2018)
Analogue C4 provides a new lead for the development of a curcumin-based compound for protection against Aβ toxicity with an improved BBB permeability.
Keyphrases
  • blood brain barrier
  • oxidative stress
  • endothelial cells
  • cerebral ischemia
  • oxide nanoparticles
  • brain injury
  • subarachnoid hemorrhage