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Inflammatory cytokine TNF-α promotes corneal endothelium apoptosis via upregulating TIPE2 transcription during corneal graft rejection.

Qun WangChao WeiLi MaXin WangLin LiQingjun ZhouWeiyun Shi
Published in: Graefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv fur klinische und experimentelle Ophthalmologie (2018)
Our study identifies the molecular mechanisms underlying the interplay of TNF-α, TIPE2, and apoptosis during allograft rejection, and it suggests that both TNF-α and TIPE2 might be potential targets for the successfully grafted corneal endothelium.
Keyphrases
  • rheumatoid arthritis
  • oxidative stress
  • optical coherence tomography
  • endoplasmic reticulum stress
  • cell cycle arrest
  • wound healing
  • nitric oxide
  • cell death
  • cataract surgery
  • genome wide
  • transcription factor