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Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signaling pathway.

Guorong WenShili DengWenfeng SongHai JinJingyu XuXuemei LiuRui XiePenghong SongBiguang Tuo
Published in: BMC microbiology (2018)
H. pylori infection downregulates duodenal epithelial cellular CFTR and SLC26A6 expressions through TGFβ-mediated P38 MAPK signaling pathway, which contributes to further elucidating the pathogenesis of H. pylori-associated duodenal ulcer.
Keyphrases
  • helicobacter pylori infection
  • signaling pathway
  • cystic fibrosis
  • transforming growth factor
  • pi k akt
  • epithelial mesenchymal transition
  • helicobacter pylori
  • induced apoptosis
  • cell proliferation