Cardiac radiotherapy transiently alters left ventricular electrical properties and induces cardiomyocyte specific ventricular substrate changes in heart failure.
Felix MehrhofJudith HuettemeisterRadu TanacliMatthias BockMarkus BögnerFelix SchoenrathVolkmar FalkDaniel ZipsGerhard HindricksJin-Hong Gerds-LiFelix HohendannerPublished in: Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology (2024)
Our study supports the notion of transiently altered cardiac conduction potentially related to structural and functional cellular changes as an underlying mechanism of cRA in patients with ventricular tachycardia.
Keyphrases
- left ventricular
- heart failure
- cardiac resynchronization therapy
- hypertrophic cardiomyopathy
- acute myocardial infarction
- mitral valve
- aortic stenosis
- left atrial
- early stage
- radiation therapy
- angiotensin ii
- atrial fibrillation
- radiation induced
- coronary artery disease
- aortic valve
- acute heart failure
- acute coronary syndrome
- percutaneous coronary intervention
- amino acid