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Gene specific effects on brain volume and cognition of TMEM106B in frontotemporal lobar degeneration.

Marijne VandeberghEliana Marisa RamosNick Corriveau-LecavalierVijay K RamananJohn KornakCarly MesterTyler KolanderDanielle BrushaberAdam M StaffaroniDaniel GeschwindAmy WolfKejal KantarciTania F GendronLeonard PetrucelliMarleen Van den BroeckSarah WynantsMatthew C BakerSergi Borrego-ÉcijaBrian ApplebySami BarmadaAndrea BozokiDavid ClarkR Ryan DarbyBradford C DickersonKimiko Domoto-ReillyJulie A FieldsDouglas R GalaskoNupur GhoshalNeill Graff-RadfordIan M GrantLawrence S HonigGing-Yuek Robin HsiungEdward D HueyDavid IrwinDavid S KnopmanJustin Y KwanGabriel C LégerIrene LitvanJoseph C MasdeuMario F MendezChiadi OnyikeBelen PascualPeter PressmanAaron RitterErik D RobersonAllison SnyderAnna Campbell SullivanM Carmela TartagliaDylan WintHilary W HeuerLeah K ForsbergAdam L BoxerHoward J RosenBradley F BoeveRosa Rademakers
Published in: medRxiv : the preprint server for health sciences (2024)
mutation carriers could additionally reflect TMEM106B's impact on divergent pathophysiological changes before the appearance of clinical symptoms.
Keyphrases
  • white matter
  • resting state
  • copy number
  • genome wide
  • multiple sclerosis
  • sleep quality
  • amyotrophic lateral sclerosis
  • dna methylation
  • depressive symptoms
  • brain injury
  • subarachnoid hemorrhage