Atze T Das

Publication Activity (10 Years)

Years Active: 2021-2024
Publications (10 Years): 10

Top Topics

Circulating Tumor Cells

Tyrosine Kinase

Top Venues

Journal of virology

Cell & bioscience

This page only lists publications with an associated author ORCID identifier.

Publications

José G Dekker, Bep Klaver, Ben Berkhout, Atze T Das
Reverse transcription of HIV-1 2-LTR circle transcripts does frequently cause 3'-polypurine tract mutations implicated in dolutegravir resistance. Journal of virology 98 (6) (2024)
José G Dekker, Bep Klaver, Ben Berkhout, Atze T Das
Reverse transcription of HIV-1 2-LTR circle transcripts does frequently cause 3'-polypurine tract mutations implicated in dolutegravir resistance. Journal of virology (2024)
José G Dekker, Bep Klaver, Ben Berkhout, Atze T Das
Reverse transcription of HIV-1 2-LTR circle transcripts does frequently cause 3'-polypurine tract mutations implicated in dolutegravir resistance. Journal of virology 98 (6) (2024)
José G Dekker, Bep Klaver, Ben Berkhout, Atze T Das
Reverse transcription of HIV-1 2-LTR circle transcripts does frequently cause 3'-polypurine tract mutations implicated in dolutegravir resistance. Journal of virology 98 (6) (2024)
Ye Liu, Caroline S Binda, Ben Berkhout, Atze T Das
CRISPR-Cas attack of HIV-1 proviral DNA can cause unintended deletion of surrounding cellular DNA. Journal of virology (2023)
José G Dekker, Bep Klaver, Ben Berkhout, Atze T Das
HIV-1 3'-Polypurine Tract Mutations Confer Dolutegravir Resistance by Switching to an Integration-Independent Replication Mechanism via 1-LTR Circles. Journal of virology (2023)
Lise Sarah Namer, Alex Harwig, Stephan P Heynen, Atze T Das, Ben Berkhout, Raymond Kaempfer
HIV co-opts a cellular antiviral mechanism, activation of stress kinase PKR by its RNA, to enable splicing of rev/tat mRNA. Cell & bioscience 13 (1) (2023)
Lise Sarah Namer, Alex Harwig, Stephan P Heynen, Atze T Das, Ben Berkhout, Raymond Kaempfer
HIV co-opts a cellular antiviral mechanism, activation of stress kinase PKR by its RNA, to enable splicing of rev/tat mRNA. Cell & bioscience 13 (1) (2023)
Lise Sarah Namer, Alex Harwig, Stephan P Heynen, Atze T Das, Ben Berkhout, Raymond Kaempfer
HIV co-opts a cellular antiviral mechanism, activation of stress kinase PKR by its RNA, to enable splicing of rev/tat mRNA. Cell & bioscience 13 (1) (2023)
Ye Liu, Rienk E Jeeninga, Bep Klaver, Ben Berkhout, Atze T Das
Transient CRISPR-Cas Treatment Can Prevent Reactivation of HIV-1 Replication in a Latently Infected T-Cell Line. Viruses 13 (12) (2021)