Daisuke Sugiura

Loop profile ORCID

Publication Activity (10 Years)

Years Active: 2019-2023
Publications (10 Years): 7

Top Topics

Bioinformatics Analysis

Immune Response

Oxidative Stress

Top Venues

The Journal of biological chemistry

Proceedings of the National Academy of Sciences of the United States of America

Science (New York, N.Y.)

This page only lists publications with an associated author ORCID identifier.

Publications

Retno Murwanti, Kaori Denda-Nagai, Daisuke Sugiura, Kaoru Mogushi, Sandra J Gendler, Tatsuro Irimura
Prevention of Inflammation-Driven Colon Carcinogenesis in Human MUC1 Transgenic Mice by Vaccination with MUC1 DNA and Dendritic Cells. Cancers 15 (6) (2023)
Retno Murwanti, Kaori Denda-Nagai, Daisuke Sugiura, Kaoru Mogushi, Sandra J Gendler, Tatsuro Irimura
Prevention of Inflammation-Driven Colon Carcinogenesis in Human MUC1 Transgenic Mice by Vaccination with MUC1 DNA and Dendritic Cells. Cancers 15 (6) (2023)
Daisuke Sugiura, Il-Mi Okazaki, Takeo K Maeda, Takumi Maruhashi, Kenji Shimizu, Rieko Arakaki, Tatsuya Takemoto, Naozumi Ishimaru, Taku Okazaki
PD-1 agonism by anti-CD80 inhibits T cell activation and alleviates autoimmunity. Nature immunology 23 (3) (2022)
Kenji Shimizu, Daisuke Sugiura, Il-Mi Okazaki, Takumi Maruhashi, Tatsuya Takemoto, Taku Okazaki
PD-1 preferentially inhibits the activation of low-affinity T cells. Proceedings of the National Academy of Sciences of the United States of America 118 (35) (2021)
Takumi Maruhashi, Daisuke Sugiura, Il-Mi Okazaki, Taku Okazaki
LAG-3: from molecular functions to clinical applications. Journal for immunotherapy of cancer 8 (2) (2021)
Daisuke Sugiura, Takumi Maruhashi, Il-Mi Okazaki, Kenji Shimizu, Takeo K Maeda, Tatsuya Takemoto, Taku Okazaki
Restriction of PD-1 function by cis-PD-L1/CD80 interactions is required for optimal T cell responses. Science (New York, N.Y.) 364 (6440) (2019)
Natsumi Maeda, Takumi Maruhashi, Daisuke Sugiura, Kenji Shimizu, Il-Mi Okazaki, Taku Okazaki
Glucocorticoids potentiate the inhibitory capacity of programmed cell death 1 by up-regulating its expression on T cells. The Journal of biological chemistry 294 (52) (2019)